You can find some evidence for further functional variances concerning medial and lateral thalamic pathways. Lesions from the ventrocaudal thalamus and somatosensory cortex generate long-Long lasting deficits from the sensory components of pain which are very similar to All those produced by lesions on the anterolateral spinal cord pathway. https://www.proleviate.com/post/conolidine-blocks-ackr3-pain-receptors-in-what-way