Metabolic alterations linked to T2D are well characterised by epidemiological and research centered studies. The pathogenesis and progression of T2D is ascribed to four mechanisms; elevated Superior glycation close solution (AGE) formation, increased polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[7]. Till just https://www.directivepublications.org/journal-of-diabetology-research/